Importantly, contrary to our hypotheses, and unlike previously published results for AN (Duncan et al., 2017; Watson et al., 2019), ED symptoms were not associated with the selected metabolic PGSs (i.e., type 2 diabetes, insulin resistance, fasting insulin, and high‐density lipoprotein [HDL] cholesterol).This pattern suggests that ED symptoms in the population and threshold EDs may be partially etiologically related (i.e., psychiatric and anthropometric origins), but that metabolic genetic factors may differentiate between symptoms and threshold EDs. This evidence concerns the gene INS and type 2 diabetes mellitus.