It should be noted that in their SD rat models of traumatic hemorrhagic shock, Masoumi et al. [18] discovered that HES resuscitation regulates the balance of TH1/TH2 response, inhibits NF-κB activation, and promotes CD4+T cell apoptosis of the traumatic hemorrhagic rats, thus improving the immunosuppression, disorder, and inflammation of the rats, which is also in line with our results. This evidence concerns the gene NFKB1 and Shock.