The extracellular signal-regulated kinase (ERK) pathway is activated by various extracellular factors, such as growth factors and hormones, to participate in cell proliferation and differentiation, stress response, etc. SDPR is a metastasis inhibitor that inhibits epithelial-mesenchymal transformation (EMT) and migration, promoting apoptosis by interacting with ERK analysis to inhibit the extracellular ERK pathway in breast tumors. This evidence concerns the gene CAVIN2 and breast neoplasm.