MUC5B and idiopathic interstitial pneumonia: Recently, Stancil et al. (2021) have demonstrated that one copy (GT) of MUC5B promoter variant prolonged the persistence of amphiregulin-induced unjammed phase in IPF-derived airway epithelial cells as compared to the no copy (GG) variant, while epithelial cells with this unjammed phase promoted proliferation and activation of human lung fibroblasts.