NFKB1 and chlamydia trachomatis infectious disease: Following chlamydial infection, the host cell response typically begins with the activation of a complex network of immune receptors (TLR2 and TR4) and their respective downstream signaling pathways (myeloid differentiation primary response 88, MyD88, and nuclear factor kappa-light-chain-enhancer of activated B cells, NFkB) (O’Connell et al., 2006; Sellami et al., 2014).