SLC1A2 and Huntington disease: The present experiments in symptomatic Q175 HET are, to the best of our knowledge, the first attempt to characterize an HD phenotype in the EAAT2 interactome by using immunoprecipitation of mYFP-tagged native and C-terminal-truncated EAAT2 as “bait.” The focus was on alterations produced by mHTT, on one side, and changes due to C-terminal truncation of EAAT2, on the other side.