Therefore, the increase in the ATP13A2 expression, as shown in this study, could be justified by two independent methods, one by binding hsa-miR-24-3p to ATP13A2 and the other by directly binding Linc01128 to ATP13A2. Therefore, the hsa-miR-24-3p and Linc01128 levels were decreased in PD; as a result, the expression of ATP13A2 was increased in this disease. This evidence concerns the gene ATP13A2 and Parkinson disease.