After contact and infection of the pulmonary epithelium, SARS-CoV-2 ends up damaging these tissues and provides the synthesis and secretion of proinflammatory cytokines, such as interleukins (ILs) 1 and 6, and tumor necrosis factor-alpha (TNF-α), among a large range of cytokines responsible for signaling infection by intracellular pathogens, such the interferons (IFNs) α and β, thus contributing strongly to the initiation of the cytokine storm [10]. This evidence concerns the gene TNF and infection.