NFKB1 and tendinitis: Previous research that outlined the necessity of Scleraxis for proper tendon survival, viability, development and healing [72,79,80] corresponds to our results that demonstrate, for the first time, that CA may be of great relevance in novel therapeutic strategies in tendinitis treatment as well as its prevention by targeting, at least in part, the tenogenic-stimulating transcription factor Scleraxis via the NF-κB signaling pathway.