Future studies to extend our present findings using a model of adult-onset acquired TLE into syndrome-specific models of epilepsy, such as pediatric genetic epileptic encephalopathies (i.e., SCN1A+/− models of Dravet syndrome) [65,66], could expand the generalizability of our current findings. The gene discussed is SCN1A; the disease is encephalopathy, progressive, early-onset, with brain edema and/or leukoencephalopathy.