For instance, incorporation of Nε-(2-(4-iodophenyl)acetyl)lysine, a motif that binds serum albumin weakly (Kd = 9.9 ± 1.7 μM), into the structure of [64/67Cu]Cu-RPS-085 (Figure 3) enhanced the tumor accumulation while facilitating the renal excretion of the PSMA-targeting agents [69]. Here, FOLH1 is linked to neoplasm.