Indeed, a study by Arisan et al. [63] revealed that miR-8066 elevates the cytokines of PRLR, CXCL6, IL-6, and IL-17 during the infection, while many other ones can mitigate the pathogenesis of COVID-19 disease via binding to the SARS-CoV-2 genome and inhibiting its post-transcriptional expression [64]. This evidence concerns the gene PRLR and infection.