These components may be responsible for GBE’s effects in the treatment of AD, which include: antioxidation, anti-inflammation, and antiapoptosis; protection against mitochondrial dysfunction, amyloidogenesis, and Aβ aggregation; the modulation of the ion homeostasis and phosphorylation of the tau protein; and even the induction of growth factors [11]. The gene discussed is MAPT; the disease is Alzheimer disease.