A large number of studies have shown that the expression of inflammatory chemokines (e.g., CXCL1, CXCL2, CXCL5, CXCL8), cytokines (e.g., IL-1β, IL-17, TNFα), or other molecules, such as the complement component C5a, G-CSF, GM-CSF, or tumor-derived oxysterols, which are involved in the formation, mobilization, and recruitment of neutrophils, was increased in patients and mice with cancer [39,40,41,42,43,44,45,46,47,48]. The gene discussed is CXCL1; the disease is neoplasm.