There were also similar proportions of CD3–CD56+ cells in the “high-density” and “low-density” populations derived from the same tumour specimen, indicating that the visible difference in growth density under the microscope at week 1 was not likely driven by CD3–CD56+ cells, but rather, uneven distribution of TILs in the primary tumour specimen [23]. The gene discussed is NCAM1; the disease is neoplasm.