Given that AHNAK knockdown caused impaired glucose tolerance and raised glucose levels in murine models, and as FGF1 has novel links with hyperglycaemic remission in mice [35], it is possible that other than the obvious cancer-related change of function, there may also be a novel link between diabetes and its management with AHNAK2, which may serve as an avenue of further research. This evidence concerns the gene FGF1 and diabetes mellitus.