Although respective phosphorylation levels were not affected, associated AMP-activated protein kinase (AMPK) expression levels acting as metabolic sensors were found to be increased in CAV1-expressing LNCaP PCa cells, particularly in CAV1 Y14F and CAV1 P132L cells, with slightly but not significantly increased levels of AMPK following RT. This evidence concerns the gene CAV1 and posterior cortical atrophy.