GLUD1 and hepatocellular carcinoma: Conversely, EGCG intervention markedly suppressed cell growth under low glucose conditions, yet AOA intervention didn ́t produce any remarkable anti-growth effect on HCC cells in the same conditions (Fig. 2D, 2E, and Supplementary Fig. S2B), suggesting that GDH1 mediated glutaminolysis played a more key role on cell growth in response to low glucose conditions.