MUC1 and Shock: In this regard, our results show AABs previously described as a function of pathology severity related to tissue and vascular damage such as MOK1 (50), antigenic proteins associated with lung damage such as MUC1 (51) including pro-inflammatory and inflammatory cytokines such as TNF, and interleukins like CXCL8 involved in the cytokine cascade and related with sepsis and septic shock (37, 52).