In this regard, our results show AABs previously described as a function of pathology severity related to tissue and vascular damage such as MOK1 (50), antigenic proteins associated with lung damage such as MUC1 (51) including pro-inflammatory and inflammatory cytokines such as TNF, and interleukins like CXCL8 involved in the cytokine cascade and related with sepsis and septic shock (37, 52). Here, CXCL8 is linked to Shock.