In agreement, a few articles have reported that the maintenance of the phosphorylated state of Drp1 Ser637 via either the administration of Kaempferol (Wu et al., 2017) or the knockout of a neuron-specific Drp1 promotor [protein phosphatase 2A regulatory domain Bβ2 (Flippo et al., 2020)] can elongate the mitochondria and alleviate the damage in mice brain infarct area and primary neurons exposed to oxygen-glucose deprivation (OGD). This evidence concerns the gene DNM1L and brain infarction.