These suggest that epilepsy-related factors (SRF, CK2Beta), miR-215-mediated translation control, transcription factor activation (STAT3), epigenetic regulation of DNA transcription (HDAC1), glutamate neurotransmitter signaling (-KEGG_AMYOTROPHIC_LATERAL_SCLEROSIS_ALS), Semaphorin/plexin signaling in neural plasticity (GO_CRANIAL_NERVE_MORPHOGENESIS and GO_PARASYMPATHETIC_NERVOUS_SYSTEM_DEVELOPMENT), and immune reaction/inflammation may correlate with neurocognition. The gene discussed is HDAC1; the disease is epilepsy.