Unfortunately, in a previous preliminary study of Srivorakun et al. [21], fetal blood analysis was performed only on four α-thalassemia genotypes with relatively small numbers of samples i.e., homozygous α0-thalassemia (n = 4), Hb H disease (n = 3), α0-thalassemia carrier (n = 11) and α+-thalassemia carrier (n = 3). This evidence concerns the gene GSTM1 and glycogen storage disease VI.