Furthermore, genes involved in β-oxidation (CRC1, POX1-3) were down-regulated in C. auris over the entire course of infection, in contrast especially to C. parapsilosis (up-regulation of ECI1, FOX2, FOX3, POT1, and PXP2 – all involved in β-oxidation), indicating differences in the utilization of fatty acids as an energy source (Figure 7(b), Table S6). Here, ECI1 is linked to infection.