Compared to FA, acute exposure to DE at 100 μg/m3 prior to intranasal administration of LAIV was shown to increase markers of eosinophil activation (eotaxin-1, p = 0.01; eosinophil cationic protein, p < 0.01) in subjects with allergic rhinitis, indicating DE may exacerbate LAIV-induced allergic inflammation [93]. The gene discussed is CCL11; the disease is allergic rhinitis.