ACTA1 and myocardial infarction: A recent study suggested that developmentally regulated brain protein (Drebrin), which binds to and increases the stability of actin filaments in neurons, is mainly expressed in myofibroblasts of mouse hearts after myocardial infarction or mouse lungs after BLM administration and promotes the expression of fibrosis-related genes, such as α-SMA and Col1A1 [27].