In line with the inflammatory hypothesis of bipolar disorder [42], we report altered levels of both proinflammatory (IL-17RB, IL-12, IL-12B) and regulatory (IL-10) cytokines and receptors, as well as chemokines (CXCL16, CCL3, CCL4, CCL20, CCL25), whereof only CCL3 was suggestively associated with medication. The gene discussed is IL17RB; the disease is bipolar disorder.