LMNB1 and adult-onset autosomal dominant demyelinating leukodystrophy: The same group created transgenic mice overexpressing Lmnb1 in different CNS cell lineages, demonstrating that lamin B1 overexpression in oligodendrocytes is sufficient for the onset of histopathological, molecular, and behavioural deficits characteristic of ADLD (not in neurons or astrocytes).