The experimental conclusions suggest that its anticachexia effect may be through reducing the expression of inflammatory factors, inhibiting the activation of the NF-κB pathway, reducing the level of ubiquitination in muscle tissue, alleviating fiber mitochondrial dysfunction, improving the malnutrition status and the energy metabolism of muscle tissue reduces the degradation of muscle and increases its synthesis (Figure 9). This evidence concerns the gene NFKB1 and malnutrition.