Similarly, another study suggested that p63+/Krt5+ basal-like lineage-negative epithelial progenitors (LNEPs) cells were activated after influenza or bleomycin injury in mice, and these Scgb1a1−/integrin α6β4+ LNEPs proliferated and migrated to injured alveoli, exhibiting differentiation potential towards mature ATII cells, and potentially contributing to the micro-honeycombing structure that characterizes progressive fibrotic lung disease [19, 40]. This evidence concerns the gene KRT5 and influenza.