In addition, univariate analyses showed no statistically significant difference in the baseline levels of apoA-I, apoA-II, apoA-IV, apoB100, apoC-I, apoC-IV, apoD, apoF, apoH, apoJ, apoM, or apo(a) between the participants who developed new-onset T2D during follow-up and the others. This evidence concerns the gene APOC1 and type 2 diabetes mellitus.