These studies have shown that there are significantly lower intracranial expressions of proteins involved in th2 and neutrophil proliferation [i.e., C-C motif chemokine 19 (CCL19), 20 (CCL20), and 23 (CCL23)] during stroke (30) and that the expression of various cytokines, chemokines, and inflammatory proteins taken at the time of thrombectomy may predict stroke severity (30–33). Here, CCL23 is linked to stroke disorder.