CCL19 and stroke disorder: These studies have shown that there are significantly lower intracranial expressions of proteins involved in th2 and neutrophil proliferation [i.e., C-C motif chemokine 19 (CCL19), 20 (CCL20), and 23 (CCL23)] during stroke (30) and that the expression of various cytokines, chemokines, and inflammatory proteins taken at the time of thrombectomy may predict stroke severity (30–33).