In their study to explore the mechanisms underlying how miR-124 could inhibit neuroinflammation, the authors found that in the MPTP-induced PD mouse model, and in LPS-treated mouse MG line BV2 cells, the expression of sequestosome1 (p62) and phosphoric acid p38 mitogen-activated protein kinase (p-p38) were significantly increased. Here, MAPK14 is linked to Parkinson disease.