It was found that the percentages of MHCII, CD40, CD80, and CD86 on surface of DCs obviously increased in the sepsis-induced ALI group when compared to the control and GTS-21-positive control mice group, and the increase in MHCII, CD40, and CD86 was reduced through GTS-21 intervention (Figures 1(b) and 1(c)). This evidence concerns the gene CD86 and Sepsis.