FOXM1 and ovarian serous adenocarcinoma: Specifically, the spliceosome-associated factor CTNNBL1, has been related to proliferation and invasion in HGSOC, at least partially through regulation of FOXM1 splicing [60], Notably, the FOXM1 signaling pathway is activated in 84% high-grade serous ovarian cancer [61] and linked to platinum resistance [62, 63].