IL36G and colitis: Interestingly, irradiated Il1f9−/− mice receiving either Il1f9+/+ or Il1f9−/− bone marrow cells were more resistant to DSS‐induced colitis than irradiated Il1f9+/+ mice receiving either wild‐type or Il1f9−/− bone marrow cells as monitored by weight loss, colon length, and HE staining of colons (Figure 1E–H), indicating that IL‐36γ in nonhematopoietic cells plays a dominant role in promoting DSS‐induced colitis.