In addition, we have recently shown that IL‐36γ and IL‐36Ra reciprocally regulate non‐small cell lung cancer progression by modulating GSH homeostasis and oxidative stress‐induced cell death.[41] Therefore, it is possible that IL‐36 cytokines exert pro‐ or anti‐tumor activities in different types of cancers by modulating distinct signaling pathways. The gene discussed is IL36G; the disease is lung cancer.