Furthermore, enrichment analysis showed that the mutated genes were involved in complex cancer signaling pathways (e.g., PI3K/AKT, WNT, and JAK/STAT pathways), biological processes (e.g., glycoprotein metabolic process, protein glycosylation, and activation of innate immune response), cellular components (e.g., extracellular matrix, Golgi lumen, and nuclear chromosome part), and molecular functions (e.g., extracellular matrix structural constituent, phosphatase binding, and protease binding). This evidence concerns the gene SOAT1 and cancer.