The use of inhibitory peptides that block circ-CUX1-EWSR1 interaction or LV-sh-circ-CUX1 significantly inhibited aerobic glycolysis, growth, and invasiveness of NB cells, suggesting that the circ-CUX1/EWSR1/MAZ axis is a therapeutic target for aerobic glycolysis and NB progression (Li H. et al., 2019). Here, MAZ is linked to neuroblastoma.