FPR1 and infection: However, the initial neutrophil chemotaxis in response to injury or infection involves the activation of G protein–coupled formyl peptide receptor (FPR) by N-formyl peptides, such as fMet-Leu-Phe (fMLF, a.k.a., fMLP), which is released either by injured tissues or by invading bacteria (de Oliveira et al., 2016; Roupé et al., 2010).