FPR1 and FPR2 are two FPR implicated in these responses, although FPR1 appears to be the primary FPR in responding to infection, as it has significantly higher affinity for bacterial formyl peptides, whereas FPR2 has a broader range of ligands than FPR1 and has been implicated in the resolution of inflammation in response to pro-resolving agonists, such as Annexin A1 (Ye et al., 2009; Jeong and Bae, 2020; Bena et al., 2012; Yazid et al., 2012; Serhan and Savill, 2005). The gene discussed is FPR2; the disease is infection.