Wilkinson and Russell suggested this difference to be the reason of the intradural sparing of GCA since vessel wall elastin is considered to be a major target of inflammation in GCA,21, 23however, vessel wall elastin may extend up to 5 mm intradurally6which might explain a variable involvement of intradural internal carotid arteries.21Several cases reporting intradural wall thickening or stenoses of vertebral arteries in GCA patients can be found.23, 24, 25It is noteworthy that using MRI imaging diagnosis of vessel wall inflammation itself can be challenging. The gene discussed is ELN; the disease is temporal arteritis.