Rather than regulation of transcription factors, translation, or stability of mRNAs involved in several diseases such as cardiac fibrosis, hepatic fibrosis, and cardiac hypertrophy in addition to helping fracture repair (Chen et al., 2019a; Jing et al., 2019; Yu et al., 2019; Wang et al., 2020a), SNHG7 is found to be overregulated in cancer tissues compared with healthy tissues in a wide variety of human malignancies, including bladder, prostate, gastric, colorectal, and pancreatic cancers (Li et al., 2018a; Zhong et al., 2018a; Cheng et al., 2019a; Han et al., 2019; Zhang et al., 2020a). Here, SNHG7 is linked to cancer.