Some evidence suggests that pericytes and microvascular smooth muscle cells express higher concentrations of SARS-CoV-2 receptor ACE2 than endothelial cells, and may play a key role in the pathogenesis of COVID-19 coagulopathy following SARS-CoV-2-mediated endothelial injury (He et al., 2020). The gene discussed is ACE2; the disease is COVID-19.