In this study, it was shown that although the level of DCX in the exercised ischemia group decreased due to the antioxidant induction, this cell increase in the nonexercise group went more towards glial cell production, and therefore, in the evaluation of the NeuN marker (shows mature neurons) decreased in the nonexercise group, as most cells went to produce astrocytes and neuroglia cells. Here, DCX is linked to ischemia.