Antiproliferative effect: bladder cancer cell death in a time- and dose-dependent manner and induced apoptosis via the activation of caspase-9 and caspase-3, migration of bladder cancer cells suppression, upregulation of E-cadherin and downregulation of N-cadherin, suppressed BFTC xenograft tumor growth. Here, CASP9 is linked to urinary bladder carcinoma.