The induction of the activation regulated chemokines TARC and PARC and the monocyte chemoattractant MCP-1 after stimulation of PBMCs with EmAg was lowest in stable and cured AE patients, which can be attributed to a suppressed or arrested proliferative growth of the metacestode larvae, possibly supported by long-term therapeutic interventions with benzimidazole. The gene discussed is CCL2; the disease is acrodermatitis enteropathica.