It is also feasible that blockade of the RAS by an ARB may have greater efficacy in the treatment of COVID‐19 because only the AT1R is blocked, therefore any Ang II formed could have anti‐inflammatory effects via its interaction with AT2R or via conversion by residual ACE2 to Ang‐(1‐7) which acts via AT2R, MasR, and MgrD (Figure 1). This evidence concerns the gene AGTR1 and COVID-19.