Notably, we found that ΔNp63Δ/Δ;KrasG12D/+ tumours had a 50% reduction in CCSP+/SPC+ cells compared to tumours from KrasG12D/+ mice, suggesting that ΔNp63 serves to maintain the proliferation of distal lung stem cell populations (Fig. 1d). This evidence concerns the gene SCGB1A1 and neoplasm.