The use of inflammatory markers to rule out bacterial co-infections has previously been investigated in a retrospective cohort study of 106 community-acquired pneumonia and 619 COVID-19 patients from the UK [14], reporting that a multivariate logistic regression model including baseline WBC and dynamics in CRP discriminated community-acquired pneumonia from COVID-19 with AUC 0.88 (95% CI 0.83–0.94). This evidence concerns the gene CRP and COVID-19.