CP and Zellweger syndrome: In a recent CP study of peroxisome-deficient cells using peroxins deletion strains of S. cerevisiae as a model system and fibroblasts from a Zellweger syndrome patient, Nübel and co-workers linked accumulation of miss-targeted peroxisomal membrane proteins into mitochondria to metabolic and morphological abnormalities of this organelle (Nuebel et al., 2021).