In the rat model of dilated cardiomyopathy, double overexpression of miR-19a and miR-20a in human-induced iPSC -MSCs was able to suppress the inflammatory response and promote the recovery of cardiac function in rats with dilated cardiomyopathy by inhibiting the secretion of TNF-α/IL-1ß (Sheu et al., 2021). This evidence concerns the gene IL1B and dilated cardiomyopathy.