Although transcriptional analysis of vitiligo-associated gene profiles indicated a significant increase in CCL20 [74], in the mouse model of vitiligo, overexpression of CCL22 could promote the recruitment and regulatory function of Tregs, accompanied by a decreased abundance of melanocyte-specific effector T cells, and resulted in alleviating depigmentation and preventing vitiligo in mice. Here, CCL22 is linked to vitiligo.